Article Sidebar
Published:
Jan 16, 2019DOI:
10.4314/njt.v38i1.31Keywords:
Article Details
Copyright belongs to the Faculty of Engineering, University of Nigeria, Nsukka, Nigeria
The contents of the articles are the sole opinion of the author(s) and not of NIJOTECH.
NIJOTECH allows open access for distribution of the published articles in any media so long as whole (not part) of articles are distributed.
A copyright and statement of originality documents will need to be filled out clearly and signed prior to publication of an accepted article. The Copyright form can be downloaded from https://www.nijotech.com/downloads/COPYRIGHT%20FORM.pdf while the Statement of Originality is in Statement of Originality.pdf (nijotech.com)
For articles that were developed from funded research, a clear acknowledgement of such support should be mentioned in the article with relevant references. Authors are expected to provide complete information on the sponsorship and intellectual property rights of the article together with all exceptions.
It is forbidden to publish the same research report in more than one journal.
Main Article Content
Modelling and simulation of myocardial infarction in the human cardiovascular system
Abstract
Modelling the physiological processes leading to myocardial infarction can help ameliorate the severity of the condition by improving early detection. Thus, the aim of this study was to model the cardiovascular system and simulate its response to myocardial infarction. Two methods were deployed for this simulation. The first method is the Computational Fluid Mechanics approach, simulated using Mathematica and the solutions of the resulting equations were obtained using Differential Transform Method. The second method is the Lumped Parameter method, simulated using MATLAB/Simulink. With Computational Fluid Mechanics, at 0% blockage within the arteries, no significant stress on the arterial wall was observed. At 10% and 50% blockage levels, a gradual increase in stress from the inlet through the entire arteries’ length was observed. 100% blockage resulted in an exponential increase in the stress. A similar output was seen with the Lumped Parameter approach. The blood flow decreases rapidly and reaches zero at a pressure of about 170mmHg. The responses of the different arteries to myocardial infarction as simulated can be applied in the early detection of heart diseases.
Keywords: myocardial infarction, stress, pressure, blood flow, arterial blockage
