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Endovascular treatment of vein of Galen aneurysmal malformation
Abstract
Vein of Galen aneurysmal malformation (VGAM) accounts for up to 30% of paediatric vascular malformations.1 As the internal cerebral veins develop, the anterior portion of the median prosencephalic vein of Markowski (MPM) regresses, and the posterior portion remains to form the vein of Galen. It is believed that the VGAM is the result of an arteriovenous connection between the primitive choroidal arteries and the MPM.2 The abnormal flow in these connections inhibits the involution of the MPM.